FREE NEJM E-TOC    HOME   |   SUBSCRIBE   |   CURRENT ISSUE   |   PAST ISSUES   |   COLLECTIONS   |   Search Term  Advanced Search

Sign in | Get NEJM's E-Mail Table of Contents — Free | Subscribe

Previous Volume 329:960-961 September 23, 1993 Number 13 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

	Full Text Purchase this article Add to Personal Archive Add to Citation Manager Notify a Friend E-mail When Cited PubMed Citation

Dilated cardiomyopathy can result from numerous underlying diseases, both intrinsic to the heart (such as coronary atherosclerosis) and extrinsic to the heart (such as catecholamine excess)¹. However, there remain many patients with dilated cardiomyopathy of unknown cause. Medical and surgical treatments for this progressive disease are palliative, and the prognosis is generally poor. Indeed, idiopathic dilated cardiomyopathy is the primary indication for heart transplantation, at a cost of approximately $177 million annually². Therefore, a better understanding of the causes and pathogenesis of the disease is urgently needed.

It is likely that idiopathic dilated cardiomyopathy has multiple causes. Recently, . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Burkett, E. L., Hershberger, R. E. (2005). Clinical and genetic issues in familial dilated cardiomyopathy. J Am Coll Cardiol 45: 969-981 [Abstract] [Full Text]  
• van den Heuvel, A. F. M., van Veldhuisen, D. J., van der Wall, E. E., Blanksma, P. K., Siebelink, H.-M. J., Vaalburg, W. M., van Gilst, W. H., Crijns, H. J. G. M. (2000). Regional myocardial blood flow reserve impairment and metabolic changes suggesting myocardial ischemia in patients with idiopathic dilated cardiomyopathy. J Am Coll Cardiol 35: 19-28 [Abstract] [Full Text]