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Previous Volume 330:1618 June 2, 1994 Number 22 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: In their Clinical Problem-Solving article (Jan. 6 issue),¹ Oren and Matzner describe a 44-year-old man who apparently had Epstein-Barr virus (EBV) hepatitis, pure red-cell aplasia, and infectious endocarditis due to Haemophilus parainfluenzae¹. Since the patient had no overt immunocompromise, the authors propose that the EBV infection was the event that precipitated the red-cell aplasia and endocarditis. They mention other possibilities, including T-cell leukemia. They invite alternative hypotheses.

Although the pathophysiologic chain of events proposed by the authors may be most reasonable, I would like to offer another hypothesis for consideration. Given the information provided, I believe . . . [Full Text of this Article]

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