FREE NEJM E-TOC    HOME   |   SUBSCRIBE   |   CURRENT ISSUE   |   PAST ISSUES   |   COLLECTIONS   |   Search Term  Advanced Search

Sign in | Get NEJM's E-Mail Table of Contents — Free | Subscribe

Previous Volume 330:1620 June 2, 1994 Number 22 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

	Full Text Purchase this article Add to Personal Archive Add to Citation Manager Notify a Friend E-mail When Cited Related Article by Parrillo, J. E.

To the Editor: Nitric oxide is a central mediator in septic shock because of its ability to relax smooth-muscle cells in arterial walls¹. High levels of nitrate, one of the metabolites of nitric oxide, can be found in the serum of hypotensive patients with sepsis^2,3. Several important features of nitric oxide in septic shock are illustrated by the case of a liver-transplant recipient who had two episodes of septic shock due to gram-negative bacteria, during which we measured blood concentrations of endotoxin, cytokines, and metabolites of nitric oxide (Table 1). A bout of septicemia due to . . . [Full Text of this Article]

References

HOME  |  SUBSCRIBE  |  SEARCH  |  CURRENT ISSUE  |  PAST ISSUES  |  COLLECTIONS  |  PRIVACY  |  TERMS OF USE  |  HELP  |  beta.nejm.org Comments and questions? Please contact us. The New England Journal of Medicine is owned, published, and copyrighted © 2009 Massachusetts Medical Society. All rights reserved.