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Previous Volume 331:130-131 July 14, 1994 Number 2 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Golino et al. (Feb. 24 issue)¹ showed that serotonin is released into the coronary circulation during angioplasty and that this vasoactive substance may contribute to the occurrence of vasoconstriction distal to the dilated site. Furthermore, the authors showed that the distal vasoconstriction is attenuated by intravenous ketanserin, a serotonin₂-receptor antagonist.

We have recently examined^2,3 the effects of the intracoronary infusion of 1 mg of ketanserin on proximal and distal coronary arterial segments in 12 patients immediately after percutaneous transluminal coronary angioplasty (PTCA), in both vessels subjected to PTCA and control vessels. After PTCA, marked vasoconstriction was . . . [Full Text of this Article]

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