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Previous Volume 332:1516-1518 June 1, 1995 Number 22 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: The report by Woods et al. (Dec. 22 issue)¹ on spreading oligemia as demonstrated by positron-emission tomography (PET) during a spontaneous episode of migraine confirms previous observations made on the basis of conventional studies of cerebral blood flow, as discussed by Olesen in the accompanying editorial.² It is a giant step, however, to conclude that this observation can be equated with "spreading depression," which is primarily an observation in experiments with animals. Besides, one could ask how spreading depression leads to the clinical features of migraine — specifically, pain, the most important symptom. What is the clinical . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Gupta, V. (2006). Silent or non-clinical infarct-like lesions in the posterior circulation territory in migraine: brain hypoperfusion or hyperperfusion?. Brain 129: E39-E39 [Full Text]