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Previous Volume 334:1341 May 16, 1996 Number 20 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Human herpesvirus 8 (HHV-8), also known as Kaposi's sarcoma–associated herpesvirus (KSHV), has recently been proposed as a possible cofactor in the development of Kaposi's sarcoma.¹ Specific sequences of the virus were detected by the polymerase chain reaction (PCR) in over 90 percent of Kaposi's sarcoma lesions, regardless of their association with the human immunodeficiency virus (HIV).² Nevertheless, except for the documented tropism of KSHV for CD19-positive circulating B lymphocytes,³ little is known about sites of viral latency and persistence in infected hosts.

In immunocompromised patients, Kaposi's sarcoma frequently presents as disseminated lesions with a varicelliform distribution (i.e., . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Chan, P. K. S., Ng, H.-K., Cheung, J. L. K., Cheng, A. F. (2000). Survey for the Presence and Distribution of Human Herpesvirus 8 in Healthy Brain. J. Clin. Microbiol. 38: 2772-2773 [Abstract] [Full Text]  
• De Milito, A., Venturi, G., Catucci, M., Romano, L., Bianchi Bandinelli, M. L., Zazzi, M. (1997). Lack of Evidence of HHV-8 DNA in Blood Cells From Heart Transplant Recipients. Blood 89: 1837-1838 [Full Text]