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Previous Volume 335:1528-1530 November 14, 1996 Number 20 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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The interaction between the human immunodeficiency virus (HIV) and its cellular target once seemed fairly simple: the virus attached to the CD4 molecule on the cell surface, entered the cell, and began its replicative cycle.¹ However, in the past few years, research has turned up surprising evidence that the virus can bind to cells by means of receptors other than CD4. These alternative receptors include galactosylceramide on brain and bowel cells and, when the virus has formed a complex with antibody, Fc receptors. Binding through Fc receptors can actually enhance viral infection.¹

Recent studies of the early events in HIV . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Levy, J.A. (2006). HIV Pathogenesis: Knowledge Gained after Two Decades of Research. Adv. Dent. Res. 19: 10-16 [Abstract] [Full Text]  
• Schwartz, S. A., Nair, M. P. N. (1999). Current Concepts in Human Immunodeficiency Virus Infection and AIDS. CVI 6: 295-305 [Full Text]