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Previous Volume 336:957-958 March 27, 1997 Number 13 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Nonautoimmune atrophic gastritis of the fundus is commonly considered chronic, progressive, and irreversible and is closely associated with gastric carcinoma and endocrine-cell tumors.^1,2 Recent investigations support the hypothesis that Helicobacter pylori may have a role in causing this condition.³

We evaluated the effect of the eradication of H. pylori on histologic findings and secretory–motor gastric functions in five men and seven women (mean [±SD] age, 48±9 years) with nonautoimmune fundic atrophic gastritis and H. pylori infection. Exclusion criteria were the presence of pernicious anemia (identified on the basis of the Schilling test, mean corpuscular volume, vitamin B₁₂ . . . [Full Text of this Article]

References

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• Iijima, K, Ohara, S, Sekine, H, Koike, T, Kato, K, Asaki, S, Shimosegawa, T, Toyota, T (2000). Changes in gastric acid secretion assayed by endoscopic gastrin test before and after Helicobacter pylori eradication. Gut 46: 20-26 [Abstract] [Full Text]  
• Kokkola, A., Rautelin, H., Puolakkainen, P., Sipponen, P., Färkkilä, M., Haapiainen, R., Kosunen, T. U. (1998). Positive Result by Serology Indicates Active Helicobacter pylori Infection in Patients with Atrophic Gastritis. J. Clin. Microbiol. 36: 1808-1810 [Abstract] [Full Text]