The New England Journal of Medicine
e-mail icon  FREE NEJM E-TOC    HOME   |   SUBSCRIBE   |   CURRENT ISSUE   |   PAST ISSUES   |   COLLECTIONS   |    Advanced Search
Sign in | Get NEJM's E-Mail Table of Contents — Free | Subscribe
 
Correspondence
PreviousPrevious
Volume 337:1555-1557 November 20, 1997 Number 21
NextNext

Autism

Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

 Sign up for free e-toc
 

This Article
-Full Text
-Purchase this article

Tools and Services
-Add to Personal Archive
-Add to Citation Manager
-Notify a Friend
-E-mail When Cited

More Information
-Related Article
 by Rapin, I.
-PubMed Citation
To the Editor: In her comprehensive review, Dr. Rapin discussed many aspects of autism (July 10 issue).1 However, we believe certain recent findings deserve greater attention than they received in the article. Evidence that abnormalities in central nervous system serotonin function have a major role in the pathophysiology of autism is compelling. It has long been known that many persons with autism have increased levels of whole-blood and platelet serotonin, accompanied by increased rates of platelet serotonin transport.2 In addition, a recent candidate-gene study revealed evidence of an association between autistic disorder and a variant of the promoter region of . . . [Full Text of this Article]

References


This article has been cited by other articles:



HOME  |  SUBSCRIBE  |  SEARCH  |  CURRENT ISSUE  |  PAST ISSUES  |  COLLECTIONS  |  PRIVACY  |  TERMS OF USE  |  HELP  |  beta.nejm.org

Comments and questions? Please contact us.

The New England Journal of Medicine is owned, published, and copyrighted © 2009 Massachusetts Medical Society. All rights reserved.