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Previous Volume 338:1061-1062 April 9, 1998 Number 15 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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Over the past decade, we have applied the power of molecular biology to the study of human diseases, and we have identified genetic defects causing over 100 different diseases. Evidence of genetic causes of osteoporosis has been with us for some time. Yet there still is considerable debate and confusion about whether osteoporosis is a genetic disease.

Anecdotal evidence indicated many years ago that blacks are much less likely to have osteoporosis than whites and Asians, apparently because of differences in genetic background.¹ In studies of monozygotic and dizygotic twins, moreover, approximately 80 percent of the age-specific variation in bone . . . [Full Text of this Article]

References

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• Eastell, R., Reid, D.M., Compston, J., Cooper, C., Fogelman, I., Francis, R.M., Hay, S.M., Hosking, D.J., Purdie, D.W., Ralston, S.H., Reeve, J., Russell, R.G.G., Stevenson, J.C. (2001). Secondary prevention of osteoporosis: when should a non-vertebral fracture be a trigger for action?. QJM 94: 575-597 [Abstract] [Full Text]  
• Eliakim, A., Scheett, T., Allmendinger, N., Brasel, J. A., Cooper, D. M. (2001). Training, muscle volume, and energy expenditure in nonobese American girls. J. Appl. Physiol. 90: 35-44 [Abstract] [Full Text]