Diseases involving the renal glomeruli are encountered frequentlyin clinical practice and are the most common causes of end-stagerenal disease worldwide. In the United States alone, glomerulardiseases accounted for 51 percent of the 305,876 cases of treatedend-stage renal disease that were reported to the U.S. RenalData System between 1991 and 1995, including 115,938 cases ofdiabetic nephropathy (37.9 percent) and 41,333 cases of nondiabeticglomerular disease (13.5 percent).1 Some common glomerular diseasesdo not cause progressive renal failure but are important causesof morbidity and sources of considerable medical expense.
From the Departments of Medicine (D.E.H., J.R.S.) and Pathology (M.C.-P.), Case Western Reserve University School of Medicine; the Division of Nephrology, University Hospitals of Cleveland (D.E.H.); and the Division of Nephrology, MetroHealth Medical Center (J.R.S.) all in Cleveland.
Address reprint requests to Dr. Hricik at the Department of Medicine, University Hospitals of Cleveland, 11100 Euclid Ave., Cleveland, OH 44106.
References
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Jaitovich, A.
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Suzuki, H., Suzuki, Y., Narita, I., Aizawa, M., Kihara, M., Yamanaka, T., Kanou, T., Tsukaguchi, H., Novak, J., Horikoshi, S., Tomino, Y.
(2008). Toll-Like Receptor 9 Affects Severity of IgA Nephropathy. J. Am. Soc. Nephrol.
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Nanez, A., Alejandro, N. F., Falahatpisheh, M. H., Kerzee, J. K., Roths, J. B., Ramos, K. S.
(2005). Disruption of glomerular cell-cell and cell-matrix interactions in hydrocarbon nephropathy. Am. J. Physiol. Renal Physiol.
289: F1291-F1303
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Wolf, M., Rose, H., Smith, R. N.
(2005). Case 28-2005 - A 42-Year-Old Man with Weight Loss, Weakness, and a Rash. NEJM
353: 1148-1157
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Wolf, D., Hochegger, K., Wolf, A. M., Rumpold, H. F., Gastl, G., Tilg, H., Mayer, G., Gunsilius, E., Rosenkranz, A. R.
(2005). CD4+CD25+ Regulatory T Cells Inhibit Experimental Anti-Glomerular Basement Membrane Glomerulonephritis in Mice. J. Am. Soc. Nephrol.
16: 1360-1370
[Abstract][Full Text]
Vinen, C S, Oliveira, D B G
(2003). Acute glomerulonephritis. Postgrad. Med. J.
79: 206-213
[Abstract][Full Text]
Anders, H.-J., Banas, B., Linde, Y., Weller, L., Cohen, C. D., Kretzler, M., Martin, S., Vielhauer, V., Schlondorff, D., Grone, H.-J.
(2003). Bacterial CpG-DNA Aggravates Immune Complex Glomerulonephritis: Role of TLR9-Mediated Expression of Chemokines and Chemokine Receptors. J. Am. Soc. Nephrol.
14: 317-326
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(2001). Chemokine and Chemokine Receptor Expression during Initiation and Resolution of Immune Complex Glomerulonephritis. J. Am. Soc. Nephrol.
12: 919-931
[Abstract][Full Text]
Madaio, M. P., Harrington, J. T.
(2001). The Diagnosis of Glomerular Diseases: Acute Glomerulonephritis and the Nephrotic Syndrome. Arch Intern Med
161: 25-34
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Powell, D. W., Mifflin, R. C., Valentich, J. D., Crowe, S. E., Saada, J. I., West, A. B.
(1999). Myofibroblasts. I. Paracrine cells important in health and disease. Am. J. Physiol. Cell Physiol.
277: C1-C19
[Abstract][Full Text]