Atherosclerosis is an inflammatory disease. Because high plasmaconcentrations of cholesterol, in particular those of low-densitylipoprotein (LDL) cholesterol, are one of the principal riskfactors for atherosclerosis,1 the process of atherogenesis hasbeen considered by many to consist largely of the accumulationof lipids within the artery wall; however, it is much more thanthat. Despite changes in lifestyle and the use of new pharmacologicapproaches to lower plasma cholesterol concentrations,2,3 cardiovasculardisease continues to be the principal cause of death in theUnited States, Europe, and much of Asia.4,5 In fact, the lesionsof atherosclerosis represent a series . . . [Full Text of this Article]
Factors That Induce and Promote Inflammation or Atherogenesis
Hypercholesterolemia and Modified Lipids and Lipoproteins
Homocysteine
Hypertension
Infection
The Nature of the Inflammatory Response
Interactions among Endothelial Cells, Monocytes, and T Cells
Monocytes and Immunity
Platelets
Atherosclerosis in Relation to Other Chronic Inflammatory Diseases
Inflammatory Response
Instability and Rupture of Plaque
New Perspectives on the Formation and Progression of Lesions
Smooth Muscle
The Role of the Matrix
Conclusions
Source Information
From the Department of Pathology, University of Washington School of Medicine, Box 357470, Seattle, WA 98195-7470, where reprint requests should be addressed to Dr. Ross (e-mail: rross@u.washington.edu).
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