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Previous Volume 340:2004-2005 June 24, 1999 Number 25 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

	Full Text Purchase this article Add to Personal Archive Add to Citation Manager Notify a Friend E-mail When Cited Related Article by Raulet, D. H. Related Article by Handgretinger, R.

To the Editor: Handgretinger et al. (Jan. 28 issue)¹ present evidence that a clone of large granular lymphocytes induced pure red-cell aplasia by attacking erythroid progenitors with a deficiency of HLA class I antigens. They show that glycophorin A+ erythroblasts normally express low levels of HLA class I antigens and that they are lysed by the clonal cells.

But the authors provide almost no data that can explain why granulocytes are unaffected in the disease. They made no attempt to lyse myeloblasts in vitro or to measure the expression of HLA class I antigens as a function of a myeloid . . . [Full Text of this Article]

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