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Previous Volume 341:210-212 July 15, 1999 Number 3 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: A woman came to the pediatric infectious-disease clinic, requesting that we evaluate her two-month-old daughter for asplenia. An older daughter had been well until she was two years of age, at which time sepsis and disseminated intravascular coagulation developed, resulting in the loss of her fingers, left leg, and right foot. During her convalescence, a diagnosis of asplenia was made on the basis of an abnormal finding on technetium-99m scanning. There was no family history of asplenia, and the parents were not related. The family was counseled that congenital asplenia could be familial.¹ In mice, splenogenesis is . . . [Full Text of this Article]

References

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• Kruetzmann, S., Rosado, M. M., Weber, H., Germing, U., Tournilhac, O., Peter, H.-H., Berner, R., Peters, A., Boehm, T., Plebani, A., Quinti, I., Carsetti, R. (2003). Human Immunoglobulin M Memory B Cells Controlling Streptococcus pneumoniae Infections Are Generated in the Spleen. JEM 197: 939-945 [Abstract] [Full Text]