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Previous Volume 342:1743-1745 June 8, 2000 Number 23 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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The importance of understanding the pathophysiologic basis of transient cerebral ischemia or ischemic stroke became clear with Fisher's original description of stroke in patients with atherothrombotic "occlusion of the internal carotid artery" in 1951.¹ Support for this concept that different arterial disorders produce ischemic stroke with definite topologic features (specifically, size and location) grew with Fisher's further pathological descriptions of lacunar and embolic stroke.

He was the first to recognize that proper therapy required a precise pathophysiologic diagnosis that included not only characterization and localization of the ischemic disease in the brain, but also knowledge of the arterial disease and . . . [Full Text of this Article]

References

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• Bittl, J. A., Hirsch, A. T. (2004). Concomitant Peripheral Arterial Disease and Coronary Artery Disease: Therapeutic Opportunities. Circulation 109: 3136-3144 [Full Text]  
• Shahar, E., Chambless, L. E., Rosamond, W. D., Boland, L. L., Ballantyne, C. M., McGovern, P. G., Sharrett, A. R. (2003). Plasma Lipid Profile and Incident Ischemic Stroke: The Atherosclerosis Risk in Communities (ARIC) Study. Stroke 34: 623-631 [Abstract] [Full Text]  
• Goldstein, L. B., Howard, G., Cohen, S. N., Toole, J. F., Tunick, P. A., Kronzon, I., Inzitari, D., Eliasziw, M., Barnett, H.J.M. (2000). Stroke in Patients with Asymptomatic Internal-Carotid-Artery Stenosis. NEJM 343: 1420-1421 [Full Text]