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Previous Volume 344:1937-1939 June 21, 2001 Number 25 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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During the past 10 years there has been a revolution in the management of acute coronary syndromes, which include unstable angina and non–Q-wave myocardial infarction, and reductions in the rate of adverse events after percutaneous coronary revascularization. Physicians today use a wealth of newly developed drug therapies, including low-molecular-weight heparins, thrombin inhibitors, and inhibitors of platelet activation.^1,2 Among these therapies, intravenous agents that block the final common pathway for platelet activation, called glycoprotein IIb/IIIa inhibitors, have been studied and approved for use in patients with acute coronary syndromes and in those undergoing percutaneous coronary revascularization. Since 1993, three glycoprotein IIb/IIIa . . . [Full Text of this Article]

References

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• Owen, A., Peverill, R. E., Moir, S., Tahiliani, N., Trivedi, A., Deshmukh, H. G., Yende, S., Yusuf, S., Mehta, S. R. (2002). Treatment of Acute Coronary Syndromes. NEJM 346: 206-208 [Full Text]  
• (2001). Are All Glycoprotein IIb/IIIa Inhibitors Equivalent?. JWatch General 2001: 3-3 [Full Text]