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Previous Volume 345:1135 October 11, 2001 Number 15 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Most cases of male pseudohermaphroditism have a genetic origin.¹ A nongenetic variant of the disorder, characterized by reduced prenatal growth and the lack of evidence of any associated malformation or endocrinopathy,² has not been established.

We report here on a pair of twins (karyotype, 46,XY) who were born at term after a normal gestation sustained by one placenta. Monozygosity was corroborated by identity for seven informative DNA loci in six chromosomes (D5S818, D13S317, D7S820, VWA, FGA, THO1, and CSF1PO). One boy (birth weight, 3.0 kg; length, 48 cm) had normal genitalia. The twin (birth weight, 1.7 kg; . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Ibanez, L., Valls, C., Cols, M., Ferrer, A., Marcos, M. V., de Zegher, F. (2002). Hypersecretion of FSH in Infant Boys and Girls Born Small for Gestational Age. J. Clin. Endocrinol. Metab. 87: 1986-1988 [Abstract] [Full Text]  
• Williams, M. S., Mendonca, B. B., Billerbeck, A. E. C., de Zegher, F. (2002). Nongenetic Male Pseudohermaphroditism. NEJM 346: 628-629 [Full Text]