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Previous Volume 346:1029-1030 March 28, 2002 Number 13 Next

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To the Editor: In discussing the case of paraneoplastic ganglionopathy with encephalopathy (Dec. 13 issue),¹ Amato and Anderson focused on the differential diagnosis and the causes of sensory ganglionopathies. Because of the primary impairment of T-shaped sensory neurons, ganglionopathies are characterized by a specific pattern of joint, muscle, and skin denervation involving myelinated and unmyelinated axons in a fashion that is not length-dependent, leading to distinct clinical, neurophysiological, and neuropathological findings. Typically, skin biopsy demonstrates similar decreases in fiber density at distal and proximal sites.² Moreover, evidence of involvement of the central sensory pathway can be used to localize the . . . [Full Text of this Article]

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