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Previous Volume 346:1331-1332 April 25, 2002 Number 17 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Mas et al. (Dec. 13 issue)¹ describe multiple potential thromboembolic mechanisms that may account for the increased risk of recurrent stroke in young patients with both patent foramen ovale and atrial septal aneurysm.

The initial descriptions of paradoxical embolism with stroke provide clear evidence of a thrombotic source (e.g., deep-vein thrombosis), evidence of thrombus movement elsewhere (pulmonary embolus, ischemia in other organs, such as the kidney, or both), and a cardiac abnormality.^2,3 Curiously, more recent articles have failed to identify an increased risk of deep-vein thrombosis, pulmonary embolism, or ischemia in other organs in patients with cardiac . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Windecker, S., Meier, B. (2008). Patent Foramen Ovale and Cryptogenic Stroke: To Close or Not to Close? Closure: What Else!. Circulation 118: 1989-1997 [Full Text]  
• Windecker, S., Wahl, A., Nedeltchev, K., Arnold, M., Schwerzmann, M., Seiler, C., Mattle, H. P., Meier, B. (2004). Comparison of medical treatment with percutaneous closure of patent foramen ovale in patients with cryptogenic stroke. J Am Coll Cardiol 44: 750-758 [Abstract] [Full Text]