The New England Journal of Medicine
e-mail icon  FREE NEJM E-TOC    HOME   |   SUBSCRIBE   |   CURRENT ISSUE   |   PAST ISSUES   |   COLLECTIONS   |    Advanced Search
Sign in | Get NEJM's E-Mail Table of Contents — Free | Subscribe
 
Correspondence
PreviousPrevious
Volume 350:2525-2526 June 10, 2004 Number 24
NextNext

Diabetic Retinopathy

Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

 Sign up for free e-toc
 

This Article
-Full Text
- PDF
-PDA Full Text
-Purchase this article

Tools and Services
-Add to Personal Archive
-Add to Citation Manager
-Notify a Friend
-E-mail When Cited
-E-mail When Letters Appear

More Information
-Related Article
 by Frank, R. N.
-PubMed Citation
To the Editor: In a review article on diabetic retinopathy (Jan. 1 issue),1 Frank stated that the mechanism for the effect of panretinal, or scatter, laser treatment on diabetic retinopathy is unclear. Two references, from 1978 and 1982, were cited to support this conclusion.

The review article ignores a substantial literature on the physiological mechanism of retinal laser treatment for proliferative diabetic retinopathy and diabetic macular edema, reviewed elsewhere.2 These works confirm that scattered laser photocoagulation increases the oxygen tension in the inner retina,3 alleviates hypoxia,4 and affects the hemodynamics of the retinal circulation.5 These studies have been carried out . . . [Full Text of this Article]


This article has been cited by other articles:



HOME  |  SUBSCRIBE  |  SEARCH  |  CURRENT ISSUE  |  PAST ISSUES  |  COLLECTIONS  |  PRIVACY  |  TERMS OF USE  |  HELP  |  beta.nejm.org

Comments and questions? Please contact us.

The New England Journal of Medicine is owned, published, and copyrighted © 2009 Massachusetts Medical Society. All rights reserved.