Acute lung injury is the clinical syndrome of rapid-onset bilateralpulmonary infiltrates and hypoxemia of noncardiac origin. Whenthe hypoxemia is severe, the condition is termed the acute respiratorydistress syndrome (ARDS).1 As archetypal examples of criticalillness requiring intensive care, advanced life support, andconsiderable health care resources, acute lung injury and ARDShave attracted substantial research interest. An extensive bodyof laboratory and clinical investigation has been amassed sincethe original description almost 40 years ago, cataloguing ouradvancing knowledge of the cause, pathophysiology, and managementof these complex and often lethal syndromes. However, estimatesof their incidence . . . [Full Text of this Article]
Source Information
From the Department of Medicine and Interdepartmental Division of Critical Care Medicine, University of Toronto, Toronto (M.S.H.); and the Clinical Research, Investigation, and Systems Modeling of Acute Illness Laboratory, Department of Critical Care Medicine, University of Pittsburgh, Pittsburgh (D.C.A.).
This article has been cited by other articles:
Matt, U., Warszawska, J. M., Bauer, M., Dietl, W., Mesteri, I., Doninger, B., Haslinger, I., Schabbauer, G., Perkmann, T., Binder, C. J., Reingruber, S., Petzelbauer, P., Knapp, S.
(2009). B{beta}15-42 Protects against Acid-induced Acute Lung Injury and Secondary Pseudomonas Pneumonia In Vivo. Am. J. Respir. Crit. Care Med.
180: 1208-1217
[Abstract][Full Text]
Rubenfeld, G. D., Herridge, M. S.
(2007). Epidemiology and Outcomes of Acute Lung Injury. Chest
131: 554-562
[Abstract][Full Text]
Previous
