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Previous Volume 353:850-851 August 25, 2005 Number 8 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Gitelman's syndrome, or congenital hypokalemic hypomagnesemic hypocalciuria with metabolic alkalosis, is widely described as a benign¹ or milder² variant of Bartter's syndrome. Despite symptoms including presyncope, vertigo, ataxia, and blurred vision, few data have been accumulated regarding formal cardiac evaluation for patients with Gitelman's syndrome.³

We report a case of Gitelman's syndrome in a patient presenting with presyncope coincident with long runs of ventricular tachycardia at 230 beats per minute that was decidedly malignant. An otherwise well 39-year-old woman taking no medications (not even thiazides) presented with recurrent presyncope. An echocardiogram and the results of a stress . . . [Full Text of this Article]

This article has been cited by other articles:

• Riveira-Munoz, E., Chang, Q., Godefroid, N., Hoenderop, J. G., Bindels, R. J., Dahan, K., Devuyst, O., for the Belgian Network for the Study of Gitelman, (2007). Transcriptional and Functional Analyses of SLC12A3 Mutations: New Clues for the Pathogenesis of Gitelman Syndrome. J. Am. Soc. Nephrol. 18: 1271-1283 [Abstract] [Full Text]