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Previous Volume 360:1911-1912 April 30, 2009 Number 18 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: In the review article on endometriosis, Bulun (Jan. 15 issue)¹ states that the presence of aromatase in endometriotic lesions appears to play an important role in the production of estradiol, and it is considered to be a key factor for endometrial proliferation.

However, Delvoux et al. recently reported the absence of aromatase activity in endometriotic lesions,² and we also did not detect aromatase protein in the glandular and stromal compartments of ectopic endometrial tissue. We recently found that what was believed to be aromatase protein was mainly endogenous biotin labeling or iron deposits.³ Using three different protocols, . . . [Full Text of this Article]

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