The fat embolism syndrome is a source of considerable morbidityafter blunt trauma and is associated with a mortality of 10to 20 percent. It has been recognized since the latter partof the 19th century1,2. Nonetheless, the pathophysiology ofthis syndrome remains incompletely understood, making the pursuitof strategies for its prevention and treatment difficult. Thereport on the fat embolism syndrome by Pell et al. in this issueof the Journal3 may provide an important direction for futureefforts.
In 90 percent of instances, the fat embolism syndrome occursafter blunt trauma complicated by long-bone fractures, but . . . [Full Text of this Article]
References
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