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Previous Volume 329:206-207 July 15, 1993 Number 3 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Rossaint et al. (Feb. 11 issue)¹ discussed the potential benefits of inhaled nitric oxide in the adult respiratory distress syndrome (ARDS). Their hypothesis is that inhalation of nitric oxide causes selective vasodilation of the pulmonary vascular bed in the ventilated lung regions, improving oxygenation and cardiac performance and reducing the accumulation of extravascular lung water. The study patients had a mild elevation of pulmonary-artery pressure that was of no apparent hemodynamic consequence, since their cardiac output was normal. Inhalation of nitric oxide reduced pulmonary-artery pressure slightly and did not change cardiac output. This confirms that its hemodynamic . . . [Full Text of this Article]

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