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Previous Volume 331:948-949 October 6, 1994 Number 14 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: In their excellent review of acute pancreatitis (April 28 issue),¹ Steinberg and Tenner address, among other aspects of the disease, the pathophysiologic features of gallstone-induced pancreatitis. They suggest that the argument about whether or not Opie's common-channel theory of 1901² explains pancreatitis in humans has not been settled. After nearly a century of research, however, the evidence that the common-channel theory is invalid appears overwhelming. The common terminal conduit between the pancreatic duct and the bile duct is, if present at all, much too short in most humans to allow for communication between the two ducts (Opie's . . . [Full Text of this Article]

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