Since the introduction 10 years ago of assays for serum thyrotropinthat are sensitive enough to distinguish between normal andlow concentrations, the measurement of serum thyrotropin hasbecome the single best test of thyroid function. The reasonis that an increase in the serum thyroxine (T4) or triiodothyronine(T3) concentration sufficient to cause clinically apparent hyperthyroidisminhibits thyrotropin secretion substantially, just as a decreasein the serum thyroid hormone concentration sufficient to causehypothyroidism increases it.
Serum thyrotropin concentrations are usually undetectable inpatients with overt hyperthyroidism. They may be detectablebut low in patients with thyrotropin . . . [Full Text of this Article]
References
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Auer, J., Berent, R., Weber, T., Eber, B.
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(2000). Single-channel activity and expression of atrial L-type Ca2+ channels in patients with latent hyperthyroidism. Am. J. Physiol. Heart Circ. Physiol.
278: H723-H730
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(1994). LOW SERUM TSH: A RISK FACTOR FOR ATRIAL FIBRILLATION?. JWatch General
1994: 2-2
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