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Previous Volume 332:1782-1783 June 29, 1995 Number 26 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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The study in this issue of the Journal by Randin et al.¹ demonstrates that alcohol is an acute pressor agent — a concept that may seem counterintuitive, since alcohol has historically been considered a vasodilator, causing cutaneous flushing and recommended as beneficial to patients with angina pectoris. On the basis of modern epidemiologic data, however, alcohol has been estimated by some to be responsible for as many as 10 percent of all cases of hypertension,² more than the number resulting from all the so-called secondary causes considered together. The first substantial evidence of an association between alcohol intake and blood . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Spaak, J., Merlocco, A. C., Soleas, G. J., Tomlinson, G., Morris, B. L., Picton, P., Notarius, C. F., Chan, C. T., Floras, J. S. (2008). Dose-related effects of red wine and alcohol on hemodynamics, sympathetic nerve activity, and arterial diameter. Am. J. Physiol. Heart Circ. Physiol. 294: H605-H612 [Abstract] [Full Text]  
• Di Gennaro, C., Barilli, A., Giuffredi, C., Gatti, C., Montanari, A., Vescovi, P. P. (2000). Sodium Sensitivity of Blood Pressure in Long-Term Detoxified Alcoholics. Hypertension 35: 869-874 [Abstract] [Full Text]  
• van de Borne, P., Mark, A. L., Montano, N., Mion, D., Somers, V. K. (1997). Effects of Alcohol on Sympathetic Activity, Hemodynamics, and Chemoreflex Sensitivity. Hypertension 29: 1278-1283 [Abstract] [Full Text]  
• York, J. L., Hirsch, J. A. (1996). Residual Pressor Effects of Chronic Alcohol in Detoxified Alcoholics. Hypertension 28: 133-138 [Abstract] [Full Text]