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Previous Volume 333:880-881 September 28, 1995 Number 13 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: Ridker and colleagues (April 6 issue)¹ demonstrated that the mutation resulting in the substitution of glutamine for arginine at position 506 in the gene coding for coagulation factor V is not associated with an increased risk of myocardial infarction or stroke but is strongly associated with the occurrence of deep venous thrombosis. This study fully confirms the results of three of four previous studies^2,3,4,5 that assessed the relation between myocardial infarction and this mutation, which is responsible for resistance to activated protein C. In the largest series, there was no statistically significant difference in the prevalence of . . . [Full Text of this Article]

References

This article has been cited by other articles:

• Juul, K., Tybjarg-Hansen, A., Steffensen, R., Kofoed, S., Jensen, G., Nordestgaard, B. G. (2002). Factor V Leiden: The Copenhagen City Heart Study and 2 meta-analyses. Blood 100: 3-10 [Abstract] [Full Text]  
• Walter, M., Reinecke, H., Breithardt, G., Assmann, G., Heinrich, J., Ridker, P. M. (1998). Factor V Leiden and Thromboembolism • Response. Circulation 97: 1426-1427 [Full Text]