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Previous Volume 333:1217-1218 November 2, 1995 Number 18 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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Gilbert's syndrome consists of chronic, mild, unconjugated hyperbilirubinemia in the absence of overt hemolysis or evidence of structural or functional liver disease.^1,2,3 The elevated serum bilirubin concentration is usually noted first in adolescence, either as an incidental finding or because of a slight yellow discoloration of the sclerae. The serum bilirubin concentration characteristically fluctuates daily, occasionally falling within the normal range but at other times, especially during fasting, physical exercise, stress, intercurrent illness, or menstruation, exceeding the upper limit of normal, sometimes by as much as three to four times. The way in which the diagnostic level is selected is . . . [Full Text of this Article]

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• Stewart, C. F., Panetta, J. C., O'Shaughnessy, M. A., Throm, S. L., Fraga, C. H., Owens, T., Liu, T., Billups, C., Rodriguez-Galindo, C., Gajjar, A., Furman, W. L., McGregor, L. M. (2007). UGT1A1 Promoter Genotype Correlates With SN-38 Pharmacokinetics, but Not Severe Toxicity in Patients Receiving Low-Dose Irinotecan. JCO 25: 2594-2600 [Abstract] [Full Text]