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Previous Volume 333:393-394 August 10, 1995 Number 6 Next

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To the Editor: Vasospasm has recently been reconsidered as a possible cause of amaurosis fugax.^1,2,3 Vasospasm was suspected in patients with recurrent episodes of visual loss that could not be attributed to an embolic source, carotid stenosis, hypercoagulability, or vasculitis, and that was not relieved by aspirin, anticoagulation, or steroids. Such patients typically have immediate cessation of amaurotic attacks when treated with the calcium-channel blockers nifedipine or verapamil. Their medical histories often included atherosclerosis, migraine, or ailments related to autoimmunity, suggesting that these factors might precipitate vasoconstriction. We report on a patient with recurrent amaurosis fugax responsive to nifedipine whose . . . [Full Text of this Article]

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