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Figure 1. A 13-year-old boy presented with increasing kyphotic deformity, back pain, and progressive paraparesis seven years after a one-year course of medical therapy for tuberculosis. He reported shock-like sensations in his legs whenever he bent forward and fatigue in his thighs and calves, which limited his activities. Physical findings included a rigid gibbus (Panel A, arrow), inability to tiptoe, hyperreflexia of the ankles with mild clonus, and normal proprioception and sensation to light touch. A magnetic resonance imaging scan (Panel C) showed that the bodies of T9 through L1 had collapsed into one mass, compressing the spinal cord . . . [Full Text of this Article] |
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