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Previous Volume 337:202-203 July 17, 1997 Number 3 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: A familial form of primary pulmonary hypertension has been described and characterized.^1,2 Its incidence in the general population ranges from 1 to 2 cases per million people, and it accounts for 6 percent of the 187 cases of primary pulmonary hypertension in the registry of the National Institutes of Health.³ The histopathologic and clinical features of the familial form of the disease are the same as those of the sporadic form. Familial primary pulmonary hypertension is inherited as an autosomal dominant trait with incomplete penetrance. We recently reported⁴ its linkage to a 27-centimorgan region on chromosome 2q31-q32 . . . [Full Text of this Article]

References

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• Barker, J. M., Yu, J., Yu, L., Wang, J., Miao, D., Bao, F., Hoffenberg, E., Nelson, J. C., Gottlieb, P. A., Rewers, M., Eisenbarth, G. S. (2005). Autoantibody "Subspecificity" in Type 1 Diabetes: Risk for organ-specific autoimmunity clusters in distinct groups. Diabetes Care 28: 850-855 [Abstract] [Full Text]  
• Barker, J. M., Ide, A., Hostetler, C., Yu, L., Miao, D., Fain, P. R., Eisenbarth, G. S., Gottlieb, P. A. (2005). Endocrine and Immunogenetic Testing in Individuals with Type 1 Diabetes and 21-Hydroxylase Autoantibodies: Addison's Disease in a High-Risk Population. J. Clin. Endocrinol. Metab. 90: 128-134 [Abstract] [Full Text]  
• Morse, J H (2003). Genetic studies of pulmonary arterial hypertension. Lupus 12: 209-212 [Abstract]