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Previous Volume 341:1141-1143 October 7, 1999 Number 15 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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In countries where parasitic diseases are prevalent, these infections cause most cases of eosinophilia. Elsewhere, the majority of patients with eosinophilia are found to have atopy or, less often, drug hypersensitivity or a skin disease. There are numerous other causes of eosinophilia, but individually they are all rare.

Eosinophilia may be primary or secondary. In patients with secondary eosinophilia, hematopoietic cells are normal and the increased production of eosinophils is a cytokine-driven reactive process. In patients with primary eosinophilia, there is an acquired abnormality of a hematopoietic stem cell. In some cases, differentiation is mainly to cells of the eosinophil . . . [Full Text of this Article]

References

Related Letters:

Hypereosinophilic Syndrome
Loughlin K. R., Leone J. L., Bain B. J.
Extract | Full Text  
N Engl J Med 2000; 342:442-443, Feb 10, 2000. Correspondence

Idiopathic Eosinophilia
Guitart J., Roufosse F., Schandené L., Cogan E., Suzuki R., Seto M., Nakaura S., Simon H.-U., Dummer R.
Extract | Full Text  
N Engl J Med 2000; 342:659-661, Mar 2, 2000. Correspondence

This article has been cited by other articles:

• Roufosse, F. E., Goldman, M., Cogan, E., Gilliland, D. G., Stone, R. M., Coutre, S. E. (2003). Hypereosinophilic Syndrome. NEJM 348: 2687-2687 [Full Text]  
• Plavsic, B. M., Newman, A. C., Reuther, W. L., Terry, J. A., Drnovsek, V. H. (2003). Peripheral Blood Eosinophilia Associated with Gastrointestinal Administration of Iodinated Contrast Media. Am. J. Roentgenol. 180: 751-753 [Abstract] [Full Text]  
• Guitart, J., Roufosse, F., Schandene, L., Cogan, E., Suzuki, R., Seto, M., Nakaura, S., Simon, H.-U., Dummer, R. (2000). Idiopathic Eosinophilia. NEJM 342: 659-661 [Full Text]  
• Loughlin, K. R., Leone, J. L., Bain, B. J. (2000). Hypereosinophilic Syndrome. NEJM 342: 442-443 [Full Text]