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Previous Volume 346:124-125 January 10, 2002 Number 2 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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Kidney stones have a lifetime incidence of up to 13 percent in North America.¹ In the United States, this disorder was responsible for an estimated 1.32 million visits to physicians in 1995 and for $1.83 billion in health care costs in 1993.² In at least 70 percent of cases, the stones consist of calcium oxalate crystals, often with calcium phosphate or sodium urate.^3,4 Kidney stones generally form only in urine that is markedly supersaturated with respect to a solid phase.⁵ Heterogeneous nucleation, in which the initial ion complex is attached to a foreign surface, occurs more often and at a . . . [Full Text of this Article]

References

Related Letters:

Prevention of Recurrent Stones in Idiopathic Hypercalciuria
Teichman J., Muldowney F.P., Bataille P., Presne C., Fournier A., Gavras I., Borghi L., Maggiore U., Bushinsky D. A.
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N Engl J Med 2002; 346:1667-1669, May 23, 2002. Correspondence

This article has been cited by other articles:

• Curhan, G. C., Willett, W. C., Knight, E. L., Stampfer, M. J. (2004). Dietary Factors and the Risk of Incident Kidney Stones in Younger Women: Nurses' Health Study II. Arch Intern Med 164: 885-891 [Abstract] [Full Text]  
• Frick, K. K., Bushinsky, D. A. (2003). Molecular Mechanisms of Primary Hypercalciuria. J. Am. Soc. Nephrol. 14: 1082-1095 [Full Text]  
• Teichman, J., Muldowney, F.P., Bataille, P., Presne, C., Fournier, A., Gavras, I., Borghi, L., Maggiore, U., Bushinsky, D. A. (2002). Prevention of Recurrent Stones in Idiopathic Hypercalciuria. NEJM 346: 1667-1669 [Full Text]