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First, the role of alcohol consumption is likely to be underestimated as a cofactor in the development of nonalcoholic fatty liver disease. As Angulo states, "a daily intake as low as 20 g [of ethanol] in females and 30 g in males may be sufficient to cause alcohol-induced liver disease." In several of the studies on nonalcoholic fatty liver disease cited in the section on epidemiologic features, excessive alcohol consumption was
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