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Volume 348:3-4 January 2, 2003 Number 1 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To quote Abraham Lincoln, "we cannot escape history." Not even in the pages of a medical journal devoted to the elucidation of the scientific basis of disease can we do so. In this issue of the Journal, Hayflick et al. (pages 33–40) report on the genetic, clinical, and radiographic delineation of Hallervorden–Spatz syndrome. Mutations in the gene encoding pantothenate kinase 2 (PANK2), a key enzyme in the biosynthesis of coenzyme A, are correlated with phenotypic features observed in both classic and atypical cases of Hallervorden–Spatz syndrome. Their report is a prototypical example of the precision of the "scalpel" . . . [Full Text of this Article]

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From the Department of Neurology and Neurosurgery and the Department of Pediatrics, McGill University, and the Division of Pediatric Neurology, Montreal Children's Hospital–McGill University Health Center — all in Montreal.

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Hallervorden and History
Leach J. P., Geiderman J. M., Shevell M.
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N Engl J Med 2003; 348:1725-1726, Apr 24, 2003. Correspondence

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