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Volume 349:1587-1589 October 23, 2003 Number 17 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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A growing understanding of the importance of the rupture of atherosclerotic plaque in the pathogenesis of coronary events has led to the identification of an expanding array of markers of plaque vulnerability.^1,2 Reports of markers of inflammation and coronary risk are appearing with increasing frequency; such markers include adhesion molecules, metalloproteinases, and as described in this issue of the Journal, myeloperoxidase (reported by Brennan et al., pages 1595–1604) and glutathione peroxidase 1 (reported by Blankenberg et al., pages 1605–1613). What should the clinician look for in these reports to determine their implications for patient care?

First, the measure should add . . . [Full Text of this Article]

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From the National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Md.

Related Letters:

Prognostic Value of Myeloperoxidase in Patients with Chest Pain
Cayley W. E. Jr., Asselbergs F. W., Cohen Tervaert J.-W., Tio R. A., Brennan M.-L., Penn M. S., Hazen S. L.
Extract | Full Text | PDF  
N Engl J Med 2004; 350:516-518, Jan 29, 2004. Correspondence

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