The New England Journal of Medicine
e-mail icon  FREE NEJM E-TOC    HOME   |   SUBSCRIBE   |   CURRENT ISSUE   |   PAST ISSUES   |   COLLECTIONS   |    Advanced Search
Sign in | Get NEJM's E-Mail Table of Contents — Free | Subscribe
 
Perspective
PreviousPrevious
Volume 352:2266-2267 June 2, 2005 Number 22
NextNext

Aging, Immunity, and the Varicella–Zoster Virus
Ann Arvin, M.D.

Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

 Sign up for free e-toc
 

This Article
-Full Text
- PDF
-PDA Full Text
-Purchase this article

Tools and Services
-Add to Personal Archive
-Add to Citation Manager
-Notify a Friend
-E-mail When Cited
-E-mail When Letters Appear

More Information
-Related Article
 by Oxman, M.N.
-PubMed Citation
Older people and their physicians are all too well acquainted with herpes zoster — commonly called shingles, from the Latin cingulum, or "girdle." Most recognize that aging creates a special vulnerability to this often severely painful skin rash caused by varicella–zoster virus (VZV). VZV is so named because varicella (chickenpox) represents the first encounter between the virus and the host, and herpes zoster represents the second. VZV persists in the sensory ganglia of the cranial nerves and the spinal dorsal-root ganglia after varicella resolves, and it may become reactivated after many decades of latency; molecular analyses of VZV DNA demonstrate . . . [Full Text of this Article]


Source Information

Dr. Arvin is a professor of pediatrics and microbiology and immunology at the Stanford University School of Medicine, Stanford, Calif.


This article has been cited by other articles:



HOME  |  SUBSCRIBE  |  SEARCH  |  CURRENT ISSUE  |  PAST ISSUES  |  COLLECTIONS  |  PRIVACY  |  HELP  |  beta.nejm.org

Comments and questions? Please contact us.

The New England Journal of Medicine is owned, published, and copyrighted © 2008 Massachusetts Medical Society. All rights reserved.