Sleep occupies a third of our lives. The cardiovascular implicationsof normal and disturbed sleep, and of sleep apnea1 in particular,have only recently gained prominence. Central sleep apnea ischaracterized by intermittent loss of respiratory drive, resultingin apnea followed by compensatory periods of hyperventilation.Obstructive sleep apnea occurs when mechanisms that maintainupper-airway tone during sleep are dysfunctional, resultingin a narrowing or collapse of the airway. Obstructive sleepapnea is often treatable by some combination of postural change,weight loss, and therapy with continuous positive airway pressure(CPAP); treatment paradigms for central sleep apnea are lessclear. . . . [Full Text of this Article]
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From the Department of Medicine, Division of Cardiovascular Diseases, Mayo Clinic, Rochester, Minn.
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