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Previous Volume 353:737-738 August 18, 2005 Number 7 Next

	Since this article has no abstract, we have provided an extract of the first 100 words of the full text and any section headings.

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To the Editor: The study by Eghbali-Fatourechi et al. (May 12 issue)¹ suggests the presence of circulating osteoblasts that potentially contribute to physiological bone formation. In addition to its association with bone modeling and remodeling, as Canalis discusses in the accompanying editorial,² calcification can be observed in blood vessels and heart valves as a common complication of atherosclerosis, diabetes, and end-stage renal disease and is generally considered to be a significant predictor of adverse clinical events. Extraosseous calcification seems to be an active, cell-mediated process within bone-like tissues with marrow, cartilaginous tissue, and osteoblast-like cells.³ Since naive nonosseous tissues contain . . . [Full Text of this Article]

This article has been cited by other articles:

• TOWLER, D. A., SHAO, J.-S., CHENG, S.-L., PINGSTERHAUS, J. M., LOEWY, A. P. (2006). Osteogenic Regulation of Vascular Calcification. Ann. N. Y. Acad. Sci. 1068: 327-333 [Abstract] [Full Text]  
• KHOSLA, S., EGHBALI-FATOURECHI, G. Z. (2006). Circulating Cells with Osteogenic Potential. Ann. N. Y. Acad. Sci. 1068: 489-497 [Abstract] [Full Text]